1 erleukin 1beta converting enzyme; 0 - interleukin 1 receptor antago
2 eaving enzime (a protease) that cuts out interleukin 1, a signalling p
3 with proinflammatory cytokines, such as interleukin 1 (IL-1) and
4 ne proteases, or caspases, related to interleukin 1-beta converting
5 ine proteases, or caspases, related to interleukin 1-beta converting
6 roinflamatory cytokines.The effect of- interleukin 1 beta converting
7 hich are cysteine proteases related to interleukin 1 beta-converting
8 ic carcinoma cell line, HR, with the interleukin 2 (IL-2) gene. St
9 (GSH), lipopolysaccharide (LPS) and interleukin 2 (IL-2). Apoptos
10 ation of cellular mRNA revealed that interleukin 2 enhances the ex
11 : Animal.. Human.. Rats. AB: Effect of interleukin 2 (IL-2) on the p
12 erative effect which was cytotoxic-free: interleukin 2 did not ind
13 s. Moreover, bcl-2 expression in the interleukin 3 (IL-3)-dependen
14 f B lymphocyte precursors deprived of interleukin 3 (IL-3). These f
15 2F-1 overexpression are tolerated in interleukin 3 (IL-3)-dependen
16 B lymphocyte precursors deprived of interleukin 3 (IL-3). These f
17 as lytic necrosis and apoptosis. RN: 0 - Interleukin-2; 57-22-7 - Vinc
18 cells by the hematopoietic cytokines interleukin 6 and granulocyte
19 lished a relation between apoptosis and interleukin 7 gene expression
20 n of nonmetastatic K-1735 cells with interleukin-1 alpha (IL-1alph
21 kines (tumour necrosis factor-alpha, interleukin-1 alpha) by isola
22 T cells, or the recombinant cytokines interleukin-6 and tumor necro
23 her the cell death-related proteases, interleukin-1 beta converting
24 f expression and nuclear localization of interleukin-1 beta convert
25 duction of tumor necrosis factor and interleukin-1 beta in both in
26 also inhibited by inhibitors of ICE (interleukin-1 beta converting
27 sp-CH2-DCB (100 microM), an inhibitor of interleukin-1 beta-conver
28 ed cells. On the other hand, DAD1 and interleukin-1 beta-converting
29 These are, first, the genes encoding the interleukin-1 beta-convert
30 is at the level of the activation of ICE-(Interleukin Converting
31 g selective peptide inhibitors of the interleukin-1beta converting
32 EGF and IFN-gamma induced caspase 1 (interleukin-1beta converting
33 wn in nude mice and in vitro, whereas interleukin-6 did not. The ma
34 ing Growth Factor beta; 96282-35-8 - interleukin-1beta-converting
35 transfected with the interleukin-2 or interleukin-7 gene. In co
36 by tumor necrosis factor (TNF)-alpha, interleukin (IL)-2, IL-4, IL-
37 y of the factors. In UM-SCV-1A cells, interleukin-10 (IL-10) and in
38 SCV-1A cells, interleukin-10 (IL-10) and interleukin-13 (IL-13) cau
39 ical significance. Two other chemokines, interleukin-8 (IL-8) and
40 s were cocultured in the presence of interleukin-2 (IL-2), with pu
41 ds encoding these proteins into a murine interleukin-3 (IL-3)-depen
42 colony-stimulating factor (G-CSF) or interleukin-6 (IL-6) and to a
43 the role of the proinflammatory cytokine interleukin-1 (IL-1) in di
44 ormal signalling; the biologic basis for interleukin-1 expression in d
45 tion of Fas, and prolonged exposure to interleukin-2 increased both
46 propriate growth factors, for example interleukin-3 or GM-CSF, cult
47 of lymphocytes transfected with the interleukin-2 or interleukin-
48 colony stimulating factor (GM-CSF) or interleukin-3 prevent apoptos
49 not be explained by an induction of interleukin-6, which is an au

 (400) ….en la activación mediante IL-2 es capaz de destruir diversas células tumorales frescas in vitro….(037)

(401) La administración de células LAK autólogas, junto con grandes dosis de IL-2, ha llevado en un estudio a una reducción de más del 50% en el tumor….(037)

(402) ….tratamiento con células destructoras activadas por linfoquinas (LAK) más IL-2 recombinante…(037)

(403)….las endotoxinas, estimulan la liberación de interleukina-1 (IL-1), que es un pirógeno endógeno….

(404) ….las células T colaboradoras estimulan su propia proliferación mediante la secreción de interleuquina-2 y activan sus células diana mediante una combinación de moléculas (031)

(405)….secreción simultánea de un factor de crecimiento denominado interleuquina-2 (IL-2) y la síntesis de rectores superficie celular que se unen a él. (031)

(406) Existen por lo menos dos subclases funcionalmente distintas de células T colaboradoras, que pueden distinguirse por las interleuquinas que segregan.

(407) La célula Th1 se activa en estos momentos y descarga interleucina-2 (IL-2). Lo que producirá la activación y proliferación de la célula T citotóxica (CTL)….(031)

(408) Algunos factores de crecimiento en particular tres interleucinas (IL-1, IL-3 e IL-6), estimulan la proliferación de células madre pluripotenciales y multipotenciales….(031)